Seacrist v. Weinberger, 75-1763

Decision Date22 April 1976
Docket NumberNo. 75-1763,75-1763
Citation538 F.2d 1054
PartiesIrene SEACRIST, Appellant, v. Caspar W. WEINBERGER, Secretary of Health, Education and Welfare, Appellee.
CourtU.S. Court of Appeals — Fourth Circuit

John Boettner, Jr., Charleston, W. Va., for appellant.

John A. Field, III, U. S. Atty., Frank E. Jolliffe, Asst. U. S. Atty., Charleston, W. Va., Stephanie W. Naidoff, Regional Atty., Margaret M. Hathaway, Asst. Regional Atty., U. S. Dept. of Health, Education and Welfare, Philadelphia, Pa., for appellee.

Before HAYNSWORTH, Chief Circuit Judge, and WINTER and CRAVEN, Circuit Judges.

CRAVEN, Circuit Judge:

This is an appeal by Ms. Seacrist from a denial of widow's benefits under the Federal Coal Mine Health and Safety Act of 1969, as amended, 30 U.S.C. § 901, et seq. She contends that her husband died from pneumoconiosis (black lung) and that, as a result, she is entitled to benefits. The Secretary denied her claim, and that determination was affirmed by the district court.

The Secretary concedes that Ms. Seacrist qualifies as a dependent widow under the Act and that she properly filed her claim. The point of dispute is whether Mr. Seacrist's death was due to pneumoconiosis. 1 30 U.S.C. § 921(a); 20 C.F.R. § 410.210(e)(2). The Administrative Law Judge found that "there is insufficient evidence for concluding the miner died of or suffered from pneumoconiosis or a chronic lung disease or that such a condition significantly contributed to his death from cardiac failure." The district court held that his determination rested on substantial evidence. We affirm.

I.

The most significant evidence of cause of death is:

(1) the clinical summary made at Laird Memorial Hospital

The patient became suddenly short of breath and deeply cyanotic while working in a mine on April 30, 1963. He was first brought home and from there to Laird Memorial Hospital where he arrived moribund and demised promptly with signs of pulmonary edema. His blood pressure before death was 220/120. During the terminal illness he has apparently not complained of chest pains. He had no history of heart or for that purpose any other disease. The clinical diagnosis was made of Acute left heart failure;

(2) the autopsy protocol giving cause of death as "cardiac failure with secondary acute pulmonary edema;"

(3) a letter from Mr. Seacrist's personal physician

In reviewing the autopsy report on Brant H. Seacrist, Sr. (Bryant Hamilton Seacrist, Sr.) the findings which were described in the lungs are basically those of acute pulmonary edema, secondary to heart failure. No where in the report is there mention of any anthracotic, or silicotic deposits.

The chest X-ray, which was submitted to Social Security office was made on 8/20/55, and in reviewing the film, I find no evidence of pneumoconiosis.

However, in spite of these X-ray and autopsy findings, it is my firm opinion that this man suffered from pneumoconiosis. I treated him from 1942 until the time of his death, and throughout the entire time of knowing him, he had frequent involvement related primarily to exposure to coal dust. It is unfortunate that the autopsy findings are not more specific. The negative X-ray, of course, is of no conclusive evidence of this man suffering from pneumoconiosis.

It is my conclusion that the evidence of acute congestive failure is most likely due to right heart strain, related directly to his occupational pneumoconiosis.

It is clear that if Ms. Seacrist is to recover, it must be under 20 C.F.R. § 410.462(b) which reads as follows:

(b) Death will be found due to a respirable disease when death is medically ascribed to a chronic dust disease, or to another chronic disease of the lung. Death will not be found due to a respirable disease where the disease reported does not suggest a reasonable possibility that death was due to pneumoconiosis. Where the evidence establishes that a deceased miner suffered from pneumoconiosis or a respirable disease and death may have been due to multiple causes, death will be found due to pneumoconiosis if it is not medically feasible to distinguish which disease caused death or specifically how much each disease contributed to causing death.

As the district judge noted, this determination is more in the nature of medical finding than a judicial judgment. For that reason, after examining briefs by the parties, we requested answers to further questions posed by the court. We were interested in the meaning of the phrase "secondary acute pulmonary edema" (1) whether it was "a chronic dust disease . . . or another chronic disease of the lung" within the meaning of the first sentence of § 401.462(b), and (2) whether "secondary" as used here meant "an independent though less significant cause of death" or that pulmonary edema resulted from cardiac failure. 2

II.

The answers to these questions and others have been received and are in total conflict. Ms. Seacrist relies almost entirely on materials contained in Hurwitz & Koulack, Pneumoconiosis: Attorney's Textbook of Medicine, Vol. 8A, Ch. 25, P 205.35(3). In response to the first inquiry, she states that "(pulmonary edema) is a lung disease which can be caused by interference with pulmonary blood circulation created by fibrosis and hyalinization induced by an irritating stimulus such as coal dust particles. Moreover, 2035(d)(ii) of the Coal Miner's Manual describes pulmonary edema as a 'cardio-pulmonary disease.' " Turning to the second question, she answers: "Clearly 'secondary' used here is in the sense of an independent cause of death. Actually both the heart failure and the pulmonary edema are the manifestations of death caused by either arteriosclerosis or fibrosis of the lungs as indicated by the autopsy report. (T)h...

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